Journal of Endotoxin Research

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Fortgesetzt als Innate Immun
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Das Journal of Endotoxin Research (kurz J Endotoxin Res) ist eine medizinische Fachzeitschrift in englischer Sprache mit einem Themenschwerpunkt im Bereich der Immunologie. Sie wurde 1994 gegründet und 2006 in Innate Immunity umbenannt. Der ursprüngliche Name bezieht sich auf Endotoxine, die meist als Zerfallsprodukte von Bakterien auftreten und im Menschen zahlreiche physiologische Reaktionen auslösen können. In der Pluspedia sind zahlreiche Themen daraus erfasst.[1][2][3][4][5][6][7][8][9][10][11][12][13][14][15][16][17][18][19][20][21][22][23][24][25][26][27][28][29][30][31][32][33][34][35][36][37][38][39][40][41][42][43][44][45][46][47][48][49][50][51][52][53][54][55][56][57][58][59][60][61][62][63][64][65][66][67][68][69][70][71][72][73][74][75][76][77][78][79][80][81][82][83][84][85][86][87][88][89][90][91][92][93][94][95][96][97][98][99][100][101][102][103][104][105][106][107][108][109][110][111][112][113][114][115][116][117][118][119][120][121][122][123][124][125][126][127][128][129][130][131][132][133][134][135][136][137][138][139][140][141][142][143][144][145][146][147][148][149][150][151][152][153][154][155][156][157][158][159][160][161][162][163][164][165][166][167][168][169][170][171][172][173][174][175][176][177][178][179][180][181][182][183][184][185][186][187][188][189][190][191][192][193][194][195][196][197][198][199][200][201][202][203][204][205][206][207][208][209][210][211][212][213][214][215][216][217][218][219][220][221][222][223][224][225][226][227][228][229][230][231][232][233][234][235][236][237][238][239][240][241][242][243][244][245][246][247][248][249][250][251][252][253][254][255][256][257][258][259][260][261][262][263][264][265][266][267][268][269][270][271][272][273][274][275][276][277][278][279][280][281][282][283][284][285][286][287][288][289][290][291][292][293][294][295][296][297][298][299][300][301][302][303][304][305][306]


1 Weblinks

2 Andere Lexika

Wikipedia kennt dieses Lemma (Journal of Endotoxin Research) vermutlich nicht.

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  • Text in der englischen Wikipedia

3 Einzelnachweise

  1. Hochspringen ↑ Bikunin suppresses expression of pro-inflammatory cytokines induced by lipopolysaccharide in neutrophils. J Endotoxin Res 13(6):369-76 (2007) PMID 18182464
  2. Hochspringen ↑ Low-dose steroid alters in vivo endotoxin-induced systemic inflammation but does not influence autonomic dysfunction. J Endotoxin Res 13(6):358-68 (2007) PMID 18182463
  3. Hochspringen ↑ Physicochemical characterization and biological activity of lipooligosaccharides and lipid A from Neisseria meningitidis. J Endotoxin Res 13(6):343-57 (2007) PMID 18182462
  4. Hochspringen ↑ Human antimicrobial proteins effectors of innate immunity. J Endotoxin Res 13(6):317-38 (2007) PMID 18182460
  5. Hochspringen ↑ The role of innate immunity in the pathogenesis of asthma: evidence for the involvement of Toll-like receptor signaling. J Endotoxin Res 13(5):305-12 (2007) PMID 17986489
  6. Hochspringen ↑ Prophylactic effect of liposomal N-acetylcysteine against LPS-induced liver injuries. J Endotoxin Res 13(5):297-304 (2007) PMID 17986488
  7. Hochspringen ↑ Differential expression of Toll-like receptor 2 (TLR2) and responses to TLR2 ligands between human and murine vascular endothelial cells. J Endotoxin Res 13(5):281-96 (2007) PMID 17986487
  8. Hochspringen ↑ From therapy to experimental model: a hundred years of endotoxin administration to human subjects. J Endotoxin Res 13(5):251-79 (2007) PMID 17986486
  9. Hochspringen ↑ IEIIS Meeting minireview: Bordetella evolution: lipid A and Toll-like receptor 4. J Endotoxin Res 13(4):243-7 (2007) PMID 17956943
  10. Hochspringen ↑ High glucose up-regulates lipopolysaccharide-stimulated inflammatory cytokine production via c-jun N-terminal kinase in the monocytic cell line THP-1. J Endotoxin Res 13(4):227-34 (2007) PMID 17956941
  11. Hochspringen ↑ Peptostreptococcus micros cell wall elicits a pro-inflammatory response in human macrophages. J Endotoxin Res 13(4):219-26 (2007) PMID 17956940
  12. Hochspringen ↑ Review: variability of host-pathogen interaction. J Endotoxin Res 13(4):199-218 (2007) PMID 17956939
  13. Hochspringen ↑ Chemical synthesis of peptidoglycan fragments for elucidation of the immunostimulating mechanism. J Endotoxin Res 13(3):189-96 (2007) PMID 17621561
  14. Hochspringen ↑ Down-regulation of TGFbeta1 and leptin ameliorates thioacetamide-induced liver injury in lipopolysaccharide-primed rats. J Endotoxin Res 13(3):176-88 (2007) PMID 17621560
  15. Hochspringen ↑ Lipopolysaccharide enhances interferon-gamma-induced nitric oxide (NO) production in murine vascular endothelial cells via augmentation of interferon regulatory factor-1 activation. J Endotoxin Res 13(3):167-75 (2007) PMID 17621559
  16. Hochspringen ↑ Effects of estrogen against LPS-induced inflammation and toxicity in primary rat glial and neuronal cultures. J Endotoxin Res 13(3):158-66 (2007) PMID 17621558
  17. Hochspringen ↑ Recombinant factor C competes against LBP to bind lipopolysaccharide and neutralizes the endotoxicity. J Endotoxin Res 13(3):150-7 (2007) PMID 17621557
  18. Hochspringen ↑ Potentiation of Toll-like receptor-induced cytokine production by (1-->3)-beta-D-glucans: implications for the monocyte activation test. J Endotoxin Res 13(3):140-9 (2007) PMID 17621556
  19. Hochspringen ↑ NOD2 and defensins: translating innate to adaptive immunity in Crohn's disease. J Endotoxin Res 13(3):135-9 (2007) PMID 17621555
  20. Hochspringen ↑ Investigation on the agonistic and antagonistic biological activities of synthetic Chlamydia lipid A and its use in in vitro enzymatic assays. J Endotoxin Res 13(2):126-32 (2007) PMID 17621554
  21. Hochspringen ↑ LPS-induced mucin expression in human sinus mucosa can be attenuated by hCLCA inhibitors. J Endotoxin Res 13(2):109-16 (2007) PMID 17621552
  22. Hochspringen ↑ Hematological indices, inflammatory markers and neutrophil CD64 expression: comparative trends during experimental human endotoxemia. J Endotoxin Res 13(2):94-100 (2007) PMID 17621550
  23. Hochspringen ↑ Transcriptional activation of MMP-13 by periodontal pathogenic LPS requires p38 MAP kinase. J Endotoxin Res 13(2):85-93 (2007) PMID 17621549
  24. Hochspringen ↑ Effects of human apolipoprotein A-I on endotoxin-induced leukocyte adhesion on endothelial cells in vivo and on the growth of Escherichia coli in vitro. J Endotoxin Res 13(1):58-64 (2007) PMID 17621547
  25. Hochspringen ↑ Inflammatory responses associated with acute coronary syndrome up-regulate IRAK-M and induce endotoxin tolerance in circulating monocytes. J Endotoxin Res 13(1):39-52 (2007) PMID 17621545
  26. Hochspringen ↑ The mRNA expression of fatty acid amide hydrolase in human whole blood correlates with sepsis. J Endotoxin Res 13(1):35-8 (2007) PMID 17621544
  27. Hochspringen ↑ Pretreatment of curcumin attenuates coagulopathy and renal injury in LPS-induced endotoxemia. J Endotoxin Res 13(1):15-23 (2007) PMID 17621542
  28. Hochspringen ↑ Pathogen-associated molecular pattern contamination as putative endogenous ligands of Toll-like receptors. J Endotoxin Res 13(1):6-14 (2007) PMID 17621541
  29. Hochspringen ↑ DNA and RNA autoantigens as autoadjuvants. J Endotoxin Res 12(6):379-84 (2006) PMID 17254393
  30. Hochspringen ↑ The molecular structure of the TLR3 extracellular domain. J Endotoxin Res 12(6):375-8 (2006) PMID 17254392
  31. Hochspringen ↑ Systemic and bronchial inflammation following LPS inhalation in asthmatic and healthy subjects. J Endotoxin Res 12(6):367-74 (2006) PMID 17254391
  32. Hochspringen ↑ Effect of endotoxin, dobutamine and dopamine on muscle mitochondrial respiration in vitro. J Endotoxin Res 12(6):358-66 (2006) PMID 17254390
  33. Hochspringen ↑ Identification of two novel LPS-binding proteins in Kupffer cells: implications in TNF-alpha production. J Endotoxin Res 12(6):352-7 (2006) PMID 17254389
  34. Hochspringen ↑ Ubiquitination and de-ubiquitination: role in regulation of signaling by Toll-like receptors. J Endotoxin Res 12(6):337-45 (2006) PMID 17254387
  35. Hochspringen ↑ Conserved and variable structural features in the lipopolysaccharide of Pseudomonas aeruginosa. J Endotoxin Res 12(6):324-36 (2006) PMID 17254386
  36. Hochspringen ↑ TLR4 agonists as immunomodulatory agents. J Endotoxin Res 12(5):313-9 (2006) PMID 17059695
  37. Hochspringen ↑ Toll-like receptor-dependent discrimination of streptococci. J Endotoxin Res 12(5):307-12 (2006) PMID 17059694
  38. Hochspringen ↑ Nitric oxide induces SOCS-1 expression in human monocytes in a TNF-alpha-dependent manner. J Endotoxin Res 12(5):296-306 (2006) PMID 17059693
  39. Hochspringen ↑ Transcriptional regulation of lipopolysaccharide (LPS)-induced Toll-like receptor (TLR) expression in murine macrophages: role of interferon regulatory factors 1 (IRF-1) and 2 (IRF-2). J Endotoxin Res 12(5):285-95 (2006) PMID 17059692
  40. Hochspringen ↑ Origin of monocytes and their differentiation to macrophages and dendritic cells. J Endotoxin Res 12(5):278-84 (2006) PMID 17059691
  41. Hochspringen ↑ Mechanisms of endotoxin neutralization by synthetic cationic compounds. J Endotoxin Res 12(5):261-77 (2006) PMID 17059690
  42. Hochspringen ↑ NALP3: a key player in caspase-1 activation. J Endotoxin Res 12(4):251-6 (2006) PMID 16953978
  43. Hochspringen ↑ The interferon response to bacterial and viral infections. J Endotoxin Res 12(4):246-50 (2006) PMID 16953977
  44. Hochspringen ↑ beta-Glucans in standardized allergen extracts. J Endotoxin Res 12(4):241-5 (2006) PMID 16953976
  45. Hochspringen ↑ TLR2- and TLR4-dependent activation of STAT1 serine phosphorylation in murine macrophages is protein kinase C-delta-independent. J Endotoxin Res 12(4):231-40 (2006) PMID 16953975
  46. Hochspringen ↑ Lipopolysaccharide-induced proliferation and adhesion of U937 cells to endothelial cells involves barium chloride sensitive hyperpolarization. J Endotoxin Res 12(4):224-30 (2006) PMID 16953974
  47. Hochspringen ↑ Diversity of endotoxin and its impact on pathogenesis. J Endotoxin Res 12(4):205-23 (2006) PMID 16953973
  48. Hochspringen ↑ Roles for accessory molecules in microbial recognition by Toll-like receptors. J Endotoxin Res 12(4):195-204 (2006) PMID 16953972
  49. Hochspringen ↑ Impaired response of VLDL lipid and apoB secretion to endotoxin in the fasted rat liver. J Endotoxin Res 12(3):181-92 (2006) PMID 16719989
  50. Hochspringen ↑ Endotoxin evaluation of eleven lipopolysaccharides by whole blood assay does not always correlate with Limulus amebocyte lysate assay. J Endotoxin Res 12(3):171-80 (2006) PMID 16719988
  51. Hochspringen ↑ Compartmentalization of the inflammatory response in sepsis and SIRS. J Endotoxin Res 12(3):151-70 (2006) PMID 16719987
  52. Hochspringen ↑ Tolerance to microbial TLR ligands: molecular mechanisms and relevance to disease. J Endotoxin Res 12(3):133-50 (2006) PMID 16719986
  53. Hochspringen ↑ Localization of the mouse defense lectin ficolin B in lysosomes of activated macrophages. J Endotoxin Res 12(2):120-6 (2006) PMID 16690015
  54. Hochspringen ↑ Inflammatory, coagulatory and circulatory responses to logarithmic increases in the endotoxin dose in the anaesthetised pig. J Endotoxin Res 12(2):99-112 (2006) PMID 16690013
  55. Hochspringen ↑ MDP and other muropeptides--direct and synergistic effects on the immune system. J Endotoxin Res 12(2):69-85 (2006) PMID 16690010
  56. Hochspringen ↑ Apoptosis induction in gastric mucous cells in vitro: lesser potency of Helicobacter pylori than Escherichia coli lipopolysaccharide, but positive interaction with ibuprofen. J Endotoxin Res 12(1):47-56 (2006) PMID 16420743
  57. Hochspringen ↑ Liver argininosuccinate synthase binds to bacterial lipopolysaccharides and lipid A and inactivates their biological activities. J Endotoxin Res 12(1):21-38 (2006) PMID 16420741
  58. Hochspringen ↑ GM-CSF priming of human monocytes is dependent on ERK1/2 activation. J Endotoxin Res 12(1):10-20 (2006) PMID 16420740
  59. Hochspringen ↑ Structural features and structural variability of the lipopolysaccharide of Yersinia pestis, the cause of plague. J Endotoxin Res 12(1):3-9 (2006) PMID 16420739
  60. Hochspringen ↑ Evidence for the requirement for CD40-CD154 interactions in resistance to infections with attenuated Salmonella. J Endotoxin Res 11(6):395-9 (2005) PMID 16303097
  61. Hochspringen ↑ How Toll-like receptors and Nod-like receptors contribute to innate immunity in mammals. J Endotoxin Res 11(6):390-4 (2005) PMID 16303096
  62. Hochspringen ↑ The role and regulation of apoptosis in sepsis. J Endotoxin Res 11(6):375-82 (2005) PMID 16303094
  63. Hochspringen ↑ Antibodies against human Toll-like receptors (TLRs): TLR distribution and localization in human dendritic cells. J Endotoxin Res 11(6):369-74 (2005) PMID 16303093
  64. Hochspringen ↑ Inhibition of TLR-4/MD-2 signaling by RP105/MD-1. J Endotoxin Res 11(6):363-8 (2005) PMID 16303092
  65. Hochspringen ↑ Role of surfactant protein D (SP-D) in innate immunity in the gastric mucosa: evidence of interaction with Helicobacter pylori lipopolysaccharide. J Endotoxin Res 11(6):357-62 (2005) PMID 16303091
  66. Hochspringen ↑ Structure/function relationships of lipoteichoic acids. J Endotoxin Res 11(6):348-56 (2005) PMID 16303090
  67. Hochspringen ↑ Synthesis of lipid A and its analogues for investigation of the structural basis for their bioactivity. J Endotoxin Res 11(6):341-7 (2005) PMID 16303089
  68. Hochspringen ↑ Mutations in TLR4 signaling that lead to increased susceptibility to infection in humans: an overview. J Endotoxin Res 11(6):333-9 (2005) PMID 16303088
  69. Hochspringen ↑ New insights into the post-translational modification of Toll-like receptor signaling molecules. J Endotoxin Res 11(6):325-32 (2005) PMID 16303087
  70. Hochspringen ↑ Reprogramming of circulatory cells in sepsis and SIRS. J Endotoxin Res 11(5):311-20 (2005) PMID 16263005
  71. Hochspringen ↑ Peptidoglycan recognition in innate immunity. J Endotoxin Res 11(5):304-10 (2005) PMID 16263004
  72. Hochspringen ↑ Endotoxin: physical requirements for cell activation. J Endotoxin Res 11(5):299-303 (2005) PMID 16263003
  73. Hochspringen ↑ Conserved mechanisms of signal transduction by Toll and Toll-like receptors. J Endotoxin Res 11(5):294-8 (2005) PMID 16263002
  74. Hochspringen ↑ Iso-osmolar prehydration shifts the cytokine response towards a more anti-inflammatory balance in human endotoxemia. J Endotoxin Res 11(5):287-93 (2005) PMID 16263001
  75. Hochspringen ↑ The response of ferritin to LPS and acute phase of Pseudomonas infection. J Endotoxin Res 11(5):267-80 (2005) PMID 16262999
  76. Hochspringen ↑ Effects of perineural capsaicin treatment of the abdominal vagus on endotoxin fever and on a non-febrile thermoregulatory event. J Endotoxin Res 11(5):260-6 (2005) PMID 16262998
  77. Hochspringen ↑ The antimicrobial properties of C-reactive protein (CRP). J Endotoxin Res 11(4):249-56 (2005) PMID 16176663
  78. Hochspringen ↑ Evolution and integration of innate immune systems from fruit flies to man: lessons and questions. J Endotoxin Res 11(4):243-8 (2005) PMID 16176662
  79. Hochspringen ↑ Non-LPS targets and actions of LPS binding protein (LBP). J Endotoxin Res 11(4):237-42 (2005) PMID 16176661
  80. Hochspringen ↑ Anti-endotoxin properties of cationic host defence peptides and proteins. J Endotoxin Res 11(4):230-6 (2005) PMID 16176660
  81. Hochspringen ↑ Modulatory effects of sCD14 and LBP on LPS-host cell interactions. J Endotoxin Res 11(4):225-9 (2005) PMID 16176659
  82. Hochspringen ↑ Heritable defects of the human TLR signalling pathways. J Endotoxin Res 11(4):220-4 (2005) PMID 16176658
  83. Hochspringen ↑ Augmentation of lipopolysaccharide-induced nitric oxide production by alpha-galactosylceramide in mouse peritoneal cells. J Endotoxin Res 11(4):213-9 (2005) PMID 16176657
  84. Hochspringen ↑ A designed TLR4/MD-2 complex to capture LPS. J Endotoxin Res 11(4):197-206 (2005) PMID 16176655
  85. Hochspringen ↑ MD-2 and Der p 2 - a tale of two cousins or distant relatives? J Endotoxin Res 11(3):186-92 (2005) PMID 15949148
  86. Hochspringen ↑ Interactions between LPS and lung surfactant proteins. J Endotoxin Res 11(3):181-5 (2005) PMID 15949147
  87. Hochspringen ↑ Role of lipid A palmitoylation in bacterial pathogenesis. J Endotoxin Res 11(3):174-80 (2005) PMID 15949146
  88. Hochspringen ↑ Lipid-mediated resistance of Gram-negative bacteria against various pore-forming antimicrobial peptides. J Endotoxin Res 11(3):167-73 (2005) PMID 15949145
  89. Hochspringen ↑ Remodeling of Helicobacter pylori lipopolysaccharide. J Endotoxin Res 11(3):161-6 (2005) PMID 15949144
  90. Hochspringen ↑ Differential maturation of murine bone-marrow derived dendritic cells with lipopolysaccharide and tumor necrosis factor-alpha. J Endotoxin Res 11(3):145-60 (2005) PMID 15949143
  91. Hochspringen ↑ Lipopolysaccharide inner core oligosaccharide structure and outer membrane stability in human pathogens belonging to the Enterobacteriaceae. J Endotoxin Res 11(3):133-44 (2005) PMID 15949142
  92. Hochspringen ↑ The role of epithelial Toll-like receptor expression in host defense and microbial tolerance. J Endotoxin Res 11(2):124-8 (2005) PMID 15949140
  93. Hochspringen ↑ Monomeric endotoxin:protein complexes are essential for TLR4-dependent cell activation. J Endotoxin Res 11(2):117-23 (2005) PMID 15949139
  94. Hochspringen ↑ Innate immunity of the human newborn: distinct cytokine responses to LPS and other Toll-like receptor agonists. J Endotoxin Res 11(2):113-6 (2005) PMID 15949138
  95. Hochspringen ↑ Structure and metabolism of peptidoglycan and molecular requirements allowing its detection by the Drosophila innate immune system. J Endotoxin Res 11(2):105-11 (2005) PMID 15949137
  96. Hochspringen ↑ Apolipoproteins modulate the inflammatory response to lipopolysaccharide. J Endotoxin Res 11(2):97-103 (2005) PMID 15949136
  97. Hochspringen ↑ Legionella pneumophila mediated activation of dendritic cells involves CD14 and TLR2. J Endotoxin Res 11(2):89-96 (2005) PMID 15949135
  98. Hochspringen ↑ Interleukin-18 does not modulate the acute-phase response. J Endotoxin Res 11(2):85-8 (2005) PMID 15949134
  99. Hochspringen ↑ Detoxifying endotoxin: time, place and person. J Endotoxin Res 11(2):69-84 (2005) PMID 15949133
  100. Hochspringen ↑ Evolution and integration of innate immune recognition systems: the Toll-like receptors. J Endotoxin Res 11(1):51-5 (2005) PMID 15826379
  101. Hochspringen ↑ Production and purification of recombinants of mouse MASP-2 and sMAP. J Endotoxin Res 11(1):47-50 (2005) PMID 15826378
  102. Hochspringen ↑ Crystal structure of a peptidoglycan recognition protein (PGRP) in complex with a muramyl tripeptide from Gram-positive bacteria. J Endotoxin Res 11(1):41-6 (2005) PMID 15826377
  103. Hochspringen ↑ Role of transforming growth factor-beta1 (TGF-beta1) in endotoxin-induced hepatic failure after extensive hepatectomy in rats. J Endotoxin Res 11(1):33-9 (2005) PMID 15826376
  104. Hochspringen ↑ Optimisation of pyrogen testing in parenterals according to different pharmacopoeias by probabilistic modelling. J Endotoxin Res 11(1):25-31 (2005) PMID 15826375
  105. Hochspringen ↑ Lipoprotein-bound endotoxin exerts an immunomodulatory effect on hepatocytes through the lipid A domain of LPS. J Endotoxin Res 11(1):19-24 (2005) PMID 15826374
  106. Hochspringen ↑ Porphyromonas gingivalis fimbriae are pro-inflammatory but do not play a prominent role in the innate immune response to P. gingivalis. J Endotoxin Res 11(1):13-8 (2005) PMID 15826373
  107. Hochspringen ↑ Endotoxin signaling in human macrophages: signaling via an alternate mechanism. J Endotoxin Res 10(6):445-52 (2004) PMID 15588429
  108. Hochspringen ↑ Deacylation and palmitoylation of lipid A by Salmonellae outer membrane enzymes modulate host signaling through Toll-like receptor 4. J Endotoxin Res 10(6):439-44 (2004) PMID 15588428
  109. Hochspringen ↑ CpG oligodeoxynucleotides stimulate IFN-gamma-inducible protein-10 production in human B cells. J Endotoxin Res 10(6):431-8 (2004) PMID 15588427
  110. Hochspringen ↑ Regulatory role of Toll-like receptor 2 during infection with Trypanosoma cruzi. J Endotoxin Res 10(6):425-30 (2004) PMID 15588426
  111. Hochspringen ↑ Use of murine embryonic fibroblasts to define Toll-like receptor activation and specificity. J Endotoxin Res 10(6):419-24 (2004) PMID 15588425
  112. Hochspringen ↑ Cell activation by Toll-like receptors: role of LBP and CD14. J Endotoxin Res 10(6):413-8 (2004) PMID 15588424
  113. Hochspringen ↑ Mechanisms of TLR9 activation. J Endotoxin Res 10(6):406-12 (2004) PMID 15588423
  114. Hochspringen ↑ p38-MAPK inhibition and endotoxin induced tubular dysfunction in men. J Endotoxin Res 10(6):402-5 (2004) PMID 15588422
  115. Hochspringen ↑ Influence of recombinant adenovirus on liver injury in endotoxicosis and its modulation by IL-10 expression. J Endotoxin Res 10(6):393-401 (2004) PMID 15588421
  116. Hochspringen ↑ 8th Biennial Conference of the International Endotoxin Society, Kyoto, Japan, November 15-18, 2004. Abstracts. J Endotoxin Res 10(5):287-388 (2004) PMID 15509416
  117. Hochspringen ↑ Ligand-dependent Toll-like receptor 4 (TLR4)-oligomerization is directly linked with TLR4-signaling. J Endotoxin Res 10(4):257-60 (2004) PMID 15373971
  118. Hochspringen ↑ Interferon response induced by Toll-like receptor signaling. J Endotoxin Res 10(4):252-6 (2004) PMID 15373970
  119. Hochspringen ↑ Comparison of CpG s-ODNs, chromatin immune complexes, and dsDNA fragment immune complexes in the TLR9-dependent activation of rheumatoid factor B cells. J Endotoxin Res 10(4):247-51 (2004) PMID 15373969
  120. Hochspringen ↑ Toll-dependent and Toll-independent immune responses in Drosophila. J Endotoxin Res 10(4):241-6 (2004) PMID 15373968
  121. Hochspringen ↑ Hemoperfusion with polymyxin B-immobilized fibers reduced the number of CD16+ CD14+ monocytes in patients with septic shock. J Endotoxin Res 10(4):229-37 (2004) PMID 15373966
  122. Hochspringen ↑ Infection, fever, and exogenous and endogenous pyrogens: some concepts have changed. J Endotoxin Res 10(4):201-22 (2004) PMID 15373964
  123. Hochspringen ↑ Bacterial endotoxin modifies heat shock factor-1 activity in RAW 264.7 cells: implications for TNF-alpha regulation during exposure to febrile range temperatures. J Endotoxin Res 10(3):175-84 (2004) PMID 15198852
  124. Hochspringen ↑ C-reactive protein: a predominant LPS-binding acute phase protein responsive to Pseudomonas infection. J Endotoxin Res 10(3):163-74 (2004) PMID 15198851
  125. Hochspringen ↑ D-galactosamine lethality model: scope and limitations. J Endotoxin Res 10(3):147-62 (2004) PMID 15198850
  126. Hochspringen ↑ LPS, dsRNA and the interferon bridge to adaptive immune responses: Trif, Tram, and other TIR adaptor proteins. J Endotoxin Res 10(2):130-6 (2004) PMID 15120005
  127. Hochspringen ↑ The nexus between systemic inflammation and disordered coagulation in sepsis. J Endotoxin Res 10(2):125-9 (2004) PMID 15120004
  128. Hochspringen ↑ TLR-independent pattern recognition receptors and anti-inflammatory mechanisms. J Endotoxin Res 10(2):120-4 (2004) PMID 15120003
  129. Hochspringen ↑ Teasing apart structural determinants of 'toxicity' and 'adjuvanticity': implications for meningococcal vaccine development. J Endotoxin Res 10(2):113-9 (2004) PMID 15120002
  130. Hochspringen ↑ Enzymology of lipid A palmitoylation in bacterial outer membranes. J Endotoxin Res 10(2):107-12 (2004) PMID 15120001
  131. Hochspringen ↑ A new method for removing endotoxin from plasma using hemocompatible affinity chromatography technology, applicable for extracorporeal treatment of septic patients. J Endotoxin Res 10(2):85-95 (2004) PMID 15119999
  132. Hochspringen ↑ Molecular mechanisms of endotoxin tolerance. J Endotoxin Res 10(2):71-84 (2004) PMID 15119998
  133. Hochspringen ↑ Endotoxin induces leukocyte transmigration and changes in permeability of the airway epithelium via protein-kinase C and extracellular regulated kinase activation. J Endotoxin Res 10(1):55-65 (2004) PMID 15025824
  134. Hochspringen ↑ Fever onset is linked to the appearance of lipopolysaccharide in the liver. J Endotoxin Res 10(1):39-53 (2004) PMID 15025823
  135. Hochspringen ↑ Characteristic biological activities of lipopolysaccharides from Sinorhizobium and Mesorhizobium. J Endotoxin Res 10(1):25-31 (2004) PMID 15025821
  136. Hochspringen ↑ Molecular cloning and characterization of mouse LITAF cDNA: role in the regulation of tumor necrosis factor-alpha (TNF-alpha) gene expression. J Endotoxin Res 10(1):15-23 (2004) PMID 15025820
  137. Hochspringen ↑ The plasma kallikrein/kinin and renin angiotensin systems in blood pressure regulation in sepsis. J Endotoxin Res 10(1):3-13 (2004) PMID 15025819
  138. Hochspringen ↑ Neural inhibition of inflammation: the cholinergic anti-inflammatory pathway. J Endotoxin Res 9(6):409-13 (2003) PMID 14733730
  139. Hochspringen ↑ Regulation of interactions of endotoxin with host cells. J Endotoxin Res 9(6):401-8 (2003) PMID 14733729
  140. Hochspringen ↑ Pseudomonas aeruginosa lipid A diversity and its recognition by Toll-like receptor 4. J Endotoxin Res 9(6):395-400 (2003) PMID 14733728
  141. Hochspringen ↑ Immunopathogenesis of Onchocerca volvulus keratitis (river blindness): a novel role for TLR4 and endosymbiotic Wolbachia bacteria. J Endotoxin Res 9(6):390-4 (2003) PMID 14733727
  142. Hochspringen ↑ An overview of clinical studies in healthy subjects and patients with severe sepsis with IC14, a CD14-specific chimeric monoclonal antibody. J Endotoxin Res 9(6):385-9 (2003) PMID 14733726
  143. Hochspringen ↑ The dual role of LBP and CD14 in response to Gram-negative bacteria or Gram-negative compounds. J Endotoxin Res 9(6):381-4 (2003) PMID 14733725
  144. Hochspringen ↑ The LPS receptor generates inflammatory signals from the cell surface. J Endotoxin Res 9(6):375-80 (2003) PMID 14733724
  145. Hochspringen ↑ Immunoneutralization of procalcitonin as therapy of sepsis. J Endotoxin Res 9(6):367-74 (2003) PMID 14733723
  146. Hochspringen ↑ Structural basis for endotoxic and antagonistic activities: investigation with novel synthetic lipid A analogs. J Endotoxin Res 9(6):361-6 (2003) PMID 14733722
  147. Hochspringen ↑ Effects of bacterial cell wall components (PAMPs) on the expression of monocyte chemoattractant protein-1 (MCP-1), macrophage inflammatory protein-1alpha (MIP-1alpha) and the chemokine receptor CCR2 by purified human blood monocytes. J Endotoxin Res 9(6):349-60 (2003) PMID 14733721
  148. Hochspringen ↑ TLR signaling at the intestinal epithelial interface. J Endotoxin Res 9(5):322-30 (2003) PMID 14577850
  149. Hochspringen ↑ Activated protein C signals through the thrombin receptor PAR1 in endothelial cells. J Endotoxin Res 9(5):317-21 (2003) PMID 14577849
  150. Hochspringen ↑ The role of STAT1 in viral sensitization to LPS. J Endotoxin Res 9(5):313-6 (2003) PMID 14577848
  151. Hochspringen ↑ Role of interferons in LPS hypersensitivity. J Endotoxin Res 9(5):308-12 (2003) PMID 14577847
  152. Hochspringen ↑ Molecular basis for lipopolysaccharide mimetic action of Taxol and flavolipin. J Endotoxin Res 9(5):301-7 (2003) PMID 14577846
  153. Hochspringen ↑ Role of lipopolysaccharide (LPS) in asthma and other pulmonary conditions. J Endotoxin Res 9(5):293-300 (2003) PMID 14577845
  154. Hochspringen ↑ Inhibition of LPS-responses by synthetic peptides derived from LBP associates with the ability of the peptides to block LBP-LPS interaction. J Endotoxin Res 9(5):281-91 (2003) PMID 14577844
  155. Hochspringen ↑ Endotoxemia and Gram-negative bacteremia as predictors of outcome in sepsis: a meta-analysis using ROC curves. J Endotoxin Res 9(5):271-9 (2003) PMID 14577843
  156. Hochspringen ↑ Mycobacterial lipoarabinomannan mediates physical interactions between TLR1 and TLR2 to induce signaling. J Endotoxin Res 9(4):264-8 (2003) PMID 12935358
  157. Hochspringen ↑ Purinergic receptor regulation of LPS-induced signaling and pathophysiology. J Endotoxin Res 9(4):256-63 (2003) PMID 12935357
  158. Hochspringen ↑ Lps2: a new locus required for responses to lipopolysaccharide, revealed by germline mutagenesis and phenotypic screening. J Endotoxin Res 9(4):250-5 (2003) PMID 12935356
  159. Hochspringen ↑ Molecular insights into the assembly and diversity of the outer core oligosaccharide in lipopolysaccharides from Escherichia coli and Salmonella. J Endotoxin Res 9(4):244-9 (2003) PMID 12935355
  160. Hochspringen ↑ Bacterial LPS and CpG DNA differentially induce gene expression profiles in mouse macrophages. J Endotoxin Res 9(4):237-43 (2003) PMID 12935354
  161. Hochspringen ↑ Differential modulation of the induction of inflammatory mediators by antibiotics in mouse macrophages in response to viable Gram-positive and Gram-negative bacteria. J Endotoxin Res 9(4):225-36 (2003) PMID 12935353
  162. Hochspringen ↑ Coagulation and inflammation. J Endotoxin Res 9(3):192-8 (2003) PMID 12831462
  163. Hochspringen ↑ IkappaB-zeta, a new anti-inflammatory nuclear protein induced by lipopolysaccharide, is a negative regulator for nuclear factor-kappaB. J Endotoxin Res 9(3):187-91 (2003) PMID 12831461
  164. Hochspringen ↑ The role of membrane-bound LBP, endotoxin aggregates, and the MaxiK channel in LPS-induced cell activation. J Endotoxin Res 9(3):181-6 (2003) PMID 12831460
  165. Hochspringen ↑ Macrophage recognition of zymosan particles. J Endotoxin Res 9(3):176-80 (2003) PMID 12831459
  166. Hochspringen ↑ TLR2 and TLR4 agonists stimulate unique repertoires of host resistance genes in murine macrophages: interferon-beta-dependent signaling in TLR4-mediated responses. J Endotoxin Res 9(3):169-75 (2003) PMID 12831458
  167. Hochspringen ↑ Identification of meningococcal LPS as a major monocyte activator in IL-10 depleted shock plasmas and CSF by blocking the CD14-TLR4 receptor complex. J Endotoxin Res 9(3):155-63 (2003) PMID 12831456
  168. Hochspringen ↑ Synergistic effects of lipopolysaccharide and interferon-gamma in inducing interleukin-8 production in human monocytic THP-1 cells is accompanied by up-regulation of CD14, Toll-like receptor 4, MD-2 and MyD88 expression. J Endotoxin Res 9(3):145-53 (2003) PMID 12831455
  169. Hochspringen ↑ The lipo-oligosaccharides of Haemophilus influenzae: an interesting array of characters. J Endotoxin Res 9(3):131-44 (2003) PMID 12831454
  170. Hochspringen ↑ Lessons from an LPS-deficient Neisseria meningitidis mutant. J Endotoxin Res 9(2):124-8 (2003) PMID 12803887
  171. Hochspringen ↑ Macrophage migration inhibitory factor (MIF) regulates host responses to endotoxin through modulation of Toll-like receptor 4 (TLR4). J Endotoxin Res 9(2):119-23 (2003) PMID 12803886
  172. Hochspringen ↑ Impact of sepsis-induced changes in plasma on LPS interactions with monocytes and plasma lipoproteins: roles of soluble CD14, LBP, and acute phase lipoproteins. J Endotoxin Res 9(2):113-8 (2003) PMID 12803885
  173. Hochspringen ↑ Differences in the mechanism of nitric oxide production between mouse vascular endothelial cells and macrophages. J Endotoxin Res 9(2):108-12 (2003) PMID 12803884
  174. Hochspringen ↑ Endotoxin tolerance: is there a clinical relevance? J Endotoxin Res 9(2):101-7 (2003) PMID 12803883
  175. Hochspringen ↑ Endotoxin boosts the vascular endothelial growth factor (VEGF) in rabbits. J Endotoxin Res 9(2):97-100 (2003) PMID 12803882
  176. Hochspringen ↑ Continuous cardiac output and hemodynamic monitoring: high temporal correlation between plasma TNF-alpha and hemodynamic changes during a sepsis-like state in cancer immunotherapy. J Endotoxin Res 9(2):91-5 (2003) PMID 12803881
  177. Hochspringen ↑ C2-ceramide inhibits LPS-induced nitric oxide production in RAW 264.7 macrophage cells through down-regulating the activation of Akt. J Endotoxin Res 9(2):85-90 (2003) PMID 12803880
  178. Hochspringen ↑ Towards antibacterial strategies: studies on the mechanisms of interaction between antibacterial peptides and model membranes. J Endotoxin Res 9(2):67-84 (2003) PMID 12803879
  179. Hochspringen ↑ Mal and MyD88: adapter proteins involved in signal transduction by Toll-like receptors. J Endotoxin Res 9(1):55-9 (2003) PMID 12691620
  180. Hochspringen ↑ Synergistic and antagonistic interactions between LPS and superantigens. J Endotoxin Res 9(1):51-4 (2003) PMID 12691619
  181. Hochspringen ↑ Lipoprotein-bound LPS induces cytokine tolerance in hepatocytes. J Endotoxin Res 9(1):45-50 (2003) PMID 12691618
  182. Hochspringen ↑ Distinct post-receptor alterations generate gene- and signal-selective adaptation and cross-adaptation of TLR4 and TLR2 in human leukocytes. J Endotoxin Res 9(1):39-44 (2003) PMID 12691617
  183. Hochspringen ↑ Association of LPS chemotype of Mannheimia (Pasteurella) haemolytica A1 with disease virulence in a model of ovine pneumonic pasteurellosis. J Endotoxin Res 9(1):25-32 (2003) PMID 12691615
  184. Hochspringen ↑ Endotoxin and the hypothalamo-pituitary-adrenal (HPA) axis. J Endotoxin Res 9(1):3-24 (2003) PMID 12691614
  185. Hochspringen ↑ Implication of Galpha i proteins and Src tyrosine kinases in endotoxin-induced signal transduction events and mediator production. J Endotoxin Res 8(6):427-35 (2002) PMID 12701623
  186. Hochspringen ↑ Antagonism of in vivo and ex vivo response to endotoxin by E5564, a synthetic lipid A analogue. J Endotoxin Res 8(6):483-8 (2002) PMID 12697095
  187. Hochspringen ↑ Modulating macrophage function with IgG immune complexes. J Endotoxin Res 8(6):477-81 (2002) PMID 12697094
  188. Hochspringen ↑ The clinical relevance of endotoxin in human sepsis: a critical analysis. J Endotoxin Res 8(6):473-6 (2002) PMID 12697093
  189. Hochspringen ↑ HMGB1 as a cytokine and therapeutic target. J Endotoxin Res 8(6):469-72 (2002) PMID 12697092
  190. Hochspringen ↑ Recognition of lipopeptides by Toll-like receptors. J Endotoxin Res 8(6):459-63 (2002) PMID 12697090
  191. Hochspringen ↑ Immunostimulatory activity of aminoalkyl glucosaminide 4-phosphates (AGPs): induction of protective innate immune responses by RC-524 and RC-529. J Endotoxin Res 8(6):453-8 (2002) PMID 12697089
  192. Hochspringen ↑ Essential role of MD-2 in B-cell responses to lipopolysaccharide and Toll-like receptor 4 distribution. J Endotoxin Res 8(6):449-52 (2002) PMID 12697088
  193. Hochspringen ↑ Direct hemoperfusion with polymyxin B-immobilized fiber improves shock and hypoxemia during endotoxemia in anesthetized sheep. J Endotoxin Res 8(6):419-26 (2002) PMID 12542853
  194. Hochspringen ↑ Antimicrobial and immunoregulatory functions of lactoferrin and its potential therapeutic application. J Endotoxin Res 8(6):403-17 (2002) PMID 12542852
  195. Hochspringen ↑ The role of the interferon regulatory factors, IRF-1 and IRF-2, in LPS-induced cyclooxygenase-2 (COX-2) expression in vivo and in vitro. J Endotoxin Res 8(5):379-88 (2002) PMID 12537697
  196. Hochspringen ↑ Lipopolysaccharide-cell interaction and induced cellular activation in whole blood of septic patients. J Endotoxin Res 8(5):371-9 (2002) PMID 12537696
  197. Hochspringen ↑ Requirement of TNF and TNF receptor type 2 for LPS-induced protection from lethal septic peritonitis. J Endotoxin Res 8(5):365-9 (2002) PMID 12537695
  198. Hochspringen ↑ Monoclonal antibodies against 3-deoxy-alpha-D-manno-oct-2-ulosonic acid (Kdo) and D-glycero-alpha-D-talo-oct-2-ulosonic acid (Ko). J Endotoxin Res 8(5):357-64 (2002) PMID 12537694
  199. Hochspringen ↑ Structural and biological characterization of highly purified hepta-acyl lipid A present in the lipopolysaccharide of the Salmonella enterica sv. Minnesota Re deep rough mutant strain R595. J Endotoxin Res 8(5):343-56 (2002) PMID 12537693
  200. Hochspringen ↑ Enhancement of endotoxin activity by muramyldipeptide. J Endotoxin Res 8(5):337-42 (2002) PMID 12537692
  201. Hochspringen ↑ LPS in microbial pathogenesis: promise and fulfilment. J Endotoxin Res 8(5):329-35 (2002) PMID 12537691
  202. Hochspringen ↑ The role of the liver in the response to LPS: experimental and clinical findings. J Endotoxin Res 8(5):319-27 (2002) PMID 12537690
  203. Hochspringen ↑ Pretreatment with troglitazone decreases lethality during endotoxemia in mice. J Endotoxin Res 8(4):307-14 (2002) PMID 12230920
  204. Hochspringen ↑ A novel strategy for the synthesis of neoglycoconjugates from deacylated deep rough lipopolysaccharides. J Endotoxin Res 8(4):295-305 (2002) PMID 12230919
  205. Hochspringen ↑ The central role of thromboxane and platelet activating factor receptors in ex vivo regulation of endotoxin-induced monocyte tissue factor activity in human whole blood. J Endotoxin Res 8(4):285-93 (2002) PMID 12230918
  206. Hochspringen ↑ Alterations in membrane-associated CD14 expression and the simultaneous liberation of soluble CD14 fragment in adherent macrophages mediated by a leukocyte carboxyl/aspartate protease. J Endotoxin Res 8(4):273-83 (2002) PMID 12230917
  207. Hochspringen ↑ Lipopolysaccharide binds to and activates A(1) adenosine receptors on human pulmonary artery endothelial cells. J Endotoxin Res 8(4):263-71 (2002) PMID 12230916
  208. Hochspringen ↑ Staphylococcal enterotoxin B induces hepatic injury and lethal shock in endotoxin-resistant C3H/HeJ mice despite a deficient macrophage response. J Endotoxin Res 8(4):253-61 (2002) PMID 12230915
  209. Hochspringen ↑ Cholinesterases from plant tissues. VI. Preliminary characterization of enzymes from Solanum melongena L. and Zea mays L. Biochim Biophys Acta 410(1):130-4 (1975) PMID 67
  210. Hochspringen ↑ Hormonal regulation of fluid reabsorption in isolated rat cauda epididymidis. Endocrinology 101(5):1391-7 (1977) PMID 21071
  211. Hochspringen ↑ Endotoxin in the environment--exposure and effects. J Endotoxin Res 8(4):241-52 (2002) PMID 12230914
  212. Hochspringen ↑ Abstracts of the 7th Conference of the International Endotoxin Society. Washington, DC, USA. 18-21 July 2002. J Endotoxin Res 8(3):143-238 (2002) PMID 12184950
  213. Hochspringen ↑ Cellular mechanism underlying LPS-induced inhibition of in vitro L-leucine transport across rabbit jejunum. J Endotoxin Res 8(2):127-33 (2002) PMID 12028752
  214. Hochspringen ↑ Investigation into the interaction of recombinant human serum albumin with Re-lipopolysaccharide and lipid A. J Endotoxin Res 8(2):115-26 (2002) PMID 12028751
  215. Hochspringen ↑ Protective effects of isohelenin, an inhibitor of nuclear factor kappaB, in endotoxic shock in rats. J Endotoxin Res 8(2):99-107 (2002) PMID 12028749
  216. Hochspringen ↑ Endotoxin tolerance-current concepts in historical perspective. J Endotoxin Res 8(2):83-98 (2002) PMID 12028748
  217. Hochspringen ↑ Endotoxin and anti-endotoxin. The contribution of the schools of Koch and Pasteur: life, milestone-experiments and concepts of Richard Pfeiffer (Berlin) and Alexandre Besredka (Paris). J Endotoxin Res 8(2):71-82 (2002) PMID 12028747
  218. Hochspringen ↑ IL-1 regulates in vivo C-X-C chemokine induction and neutrophil sequestration following endotoxemia. J Endotoxin Res 8(1):59-67 (2002) PMID 11981446
  219. Hochspringen ↑ The biological activity of a liposomal complete core lipopolysaccharide vaccine. J Endotoxin Res 8(1):39-46 (2002) PMID 11981444
  220. Hochspringen ↑ Endotoxin and anti-endotoxin. The contribution of the schools of Koch and Pasteur: life, milestone-experiments and concepts of Richard Pfeiffer (Berlin) and Alexandre Besredka (Paris). J Endotoxin Res 8(1):3-16 (2002) PMID 11981441
  221. Hochspringen ↑ Pathophysiological relevance of the CD14 receptor in surgical patients: biological activity of endotoxin is regulated by the CD14 receptor. J Endotoxin Res 7(6):461-6 (2001) PMID 11753218
  222. Hochspringen ↑ Macrophage migration inhibitory factor (MIF) modulates innate immune responses induced by endotoxin and Gram-negative bacteria. J Endotoxin Res 7(6):456-60 (2001) PMID 11753217
  223. Hochspringen ↑ Complement-dependent platelet degradation and anaphylactoid shock in mice induced by lipopolysaccharide carrying the mannose homopolymer. J Endotoxin Res 7(6):451-5 (2001) PMID 11753216
  224. Hochspringen ↑ LBP, CD14, TLR4 and the murine innate immune response to a peritoneal Salmonella infection. J Endotoxin Res 7(6):447-50 (2001) PMID 11753215
  225. Hochspringen ↑ CD14 and LBP in endotoxemia and infections caused by Gram-negative bacteria. J Endotoxin Res 7(6):439-41 (2001) PMID 11753213
  226. Hochspringen ↑ The inhibitory action of quercetin on lipopolysaccharide-induced nitric oxide production in RAW 264.7 macrophage cells. J Endotoxin Res 7(6):431-8 (2001) PMID 11753212
  227. Hochspringen ↑ Differential clearance and induction of host responses by various administered or released lipopolysaccharides. J Endotoxin Res 7(6):421-9 (2001) PMID 11753211
  228. Hochspringen ↑ Neisseria meningitidis lipopolysaccharides in human pathology. J Endotoxin Res 7(6):401-20 (2001) PMID 11753210
  229. Hochspringen ↑ The role of TLR4 in endotoxin responsiveness in humans. J Endotoxin Res 7(5):389-93 (2001) PMID 11753209
  230. Hochspringen ↑ Human models of innate immunity: local and systemic inflammatory responses. J Endotoxin Res 7(5):385-8 (2001) PMID 11753208
  231. Hochspringen ↑ Scavenger receptor-like receptors for the binding of lipopolysaccharide and lipoteichoic acid to liver endothelial and Kupffer cells. J Endotoxin Res 7(5):381-4 (2001) PMID 11753207
  232. Hochspringen ↑ Genetic determinants of lipopolysaccharide and D-galactosamine-mediated hepatocellular apoptosis and lethality. J Endotoxin Res 7(5):375-80 (2001) PMID 11753206
  233. Hochspringen ↑ The hepatocyte as a microbial product-responsive cell. J Endotoxin Res 7(5):365-73 (2001) PMID 11753205
  234. Hochspringen ↑ Vagotomy attenuates the effect of lipopolysaccharide on body temperature of rats in a dose-dependent manner. J Endotoxin Res 7(5):359-64 (2001) PMID 11753204
  235. Hochspringen ↑ Lipopolysaccharide recognition, internalisation, signalling and other cellular effects. J Endotoxin Res 7(5):335-48 (2001) PMID 11753202
  236. Hochspringen ↑ The crucial role of systemic responses in the innate (non-adaptive) host defense. J Endotoxin Res 7(4):327-32 (2001) PMID 11717591
  237. Hochspringen ↑ The role of immune activation in endotoxin-induced atherogenesis. J Endotoxin Res 7(4):322-6 (2001) PMID 11717590
  238. Hochspringen ↑ Coagulation and inflammation. J Endotoxin Res 7(4):301-4 (2001) PMID 11717589
  239. Hochspringen ↑ IC14, a CD14 specific monoclonal antibody, is a potential treatment for patients with severe sepsis. J Endotoxin Res 7(4):310-4 (2001) PMID 11717588
  240. Hochspringen ↑ Replacement therapy with hydrocortisone in catecholamine-dependent septic shock. J Endotoxin Res 7(4):305-9 (2001) PMID 11717587
  241. Hochspringen ↑ Dual roles for HMGB1: DNA binding and cytokine. J Endotoxin Res 7(4):315-21 (2001) PMID 11717586
  242. Hochspringen ↑ Toll-like receptors (TLRs) signalling and expression pattern. J Endotoxin Res 7(4):297-300 (2001) PMID 11717585
  243. Hochspringen ↑ Pan-caspase inhibitor zVAD enhances cell death in RAW246.7 macrophages. J Endotoxin Res 7(4):292-6 (2001) PMID 11717584
  244. Hochspringen ↑ Toll-like receptor-2 transduces signals for NF-kappa B activation, apoptosis and reactive oxygen species production. J Endotoxin Res 7(4):287-91 (2001) PMID 11717583
  245. Hochspringen ↑ Moesin: a potential LPS receptor on human monocytes. J Endotoxin Res 7(4):281-6 (2001) PMID 11717582
  246. Hochspringen ↑ Identification of Toll-like receptor 4 (Tlr4) as the sole conduit for LPS signal transduction: genetic and evolutionary studies. J Endotoxin Res 7(4):277-80 (2001) PMID 11717581
  247. Hochspringen ↑ Characterization of the physiological substrate for lipopolysaccharide heptosyltransferases I and II. J Endotoxin Res 7(4):263-70 (2001) PMID 11717579
  248. Hochspringen ↑ The molecular genetics and role in infection of lipopolysaccharide biosynthesis in the Bordetellae. J Endotoxin Res 7(4):251-61 (2001) PMID 11717578
  249. Hochspringen ↑ A K+ channel is involved in LPS signaling. J Endotoxin Res 7(3):243-7 (2001) PMID 11581578
  250. Hochspringen ↑ Signal integration in lipopolysaccharide (LPS)-stimulated murine macrophages. J Endotoxin Res 7(3):237-41 (2001) PMID 11581577
  251. Hochspringen ↑ Decay-accelerating factor (DAF/CD55) is a functional active element of the LPS receptor complex. J Endotoxin Res 7(3):227-31 (2001) PMID 11581575
  252. Hochspringen ↑ The expression of membrane-bound CD14 renders mouse B-1 cells susceptible to LPS. J Endotoxin Res 7(3):223-6 (2001) PMID 11581574
  253. Hochspringen ↑ The p50-homodimer mechanism in tolerance to LPS. J Endotoxin Res 7(3):219-22 (2001) PMID 11581573
  254. Hochspringen ↑ Up-regulation of gastric mucosal inflammatory responses to Helicobacter pylori lipopolysaccharide by aspirin but not indomethacin. J Endotoxin Res 7(3):203-9 (2001) PMID 11581571
  255. Hochspringen ↑ Bacterial lipopolysaccharides and innate immunity. J Endotoxin Res 7(3):167-202 (2001) PMID 11581570
  256. Hochspringen ↑ Influence of acyl chain fluidity on the lipopolysaccharide-induced activation of complement. J Endotoxin Res 7(2):147-55 (2001) PMID 11521095
  257. Hochspringen ↑ Structural and biological characterisation of a novel tetra-acyl lipid A from Escherichia coli F515 lipopolysaccharide acting as endotoxin antagonist in human monocytes. J Endotoxin Res 7(2):133-46 (2001) PMID 11521094
  258. Hochspringen ↑ Synthesis and immunochemical characterization of neoglycoproteins containing epitopes of the inner core region of Pseudomonas aeruginosa RNA group I lipopolysaccharide. J Endotoxin Res 7(2):125-31 (2001) PMID 11521093
  259. Hochspringen ↑ Structural and serological characterisation of the O-antigenic polysaccharide of the lipopolysaccharide from Acinetobacter strain 96 (DNA group 11). J Endotoxin Res 7(2):113-8 (2001) PMID 11521091
  260. Hochspringen ↑ Cytokine synthesis in the liver of endotoxin-tolerant and normal rats during hemorrhagic shock. J Endotoxin Res 7(2):105-12 (2001) PMID 11521090
  261. Hochspringen ↑ Immunodepression in sepsis and SIRS assessed by ex vivo cytokine production is not a generalized phenomenon: a review. J Endotoxin Res 7(2):85-93 (2001) PMID 11521088
  262. Hochspringen ↑ Regulated covalent modifications of lipid A. J Endotoxin Res 7(1):73-8 (2001) PMID 11521087
  263. Hochspringen ↑ Outer membrane protein A (OmpA), peptidoglycan-associated lipoprotein (PAL), and murein lipoprotein (MLP) are released in experimental Gram-negative sepsis. J Endotoxin Res 7(1):69-72 (2001) PMID 11521086
  264. Hochspringen ↑ Structural variability and originality of the Bordetella endotoxins. J Endotoxin Res 7(1):63-8 (2001) PMID 11521085
  265. Hochspringen ↑ Bacterial modification of LPS and resistance to antimicrobial peptides. J Endotoxin Res 7(1):57-62 (2001) PMID 11521084
  266. Hochspringen ↑ The involvement of Ran GTPase in lipopolysaccharide endotoxin-induced responses. J Endotoxin Res 7(1):53-6 (2001) PMID 11521083
  267. Hochspringen ↑ CD14: a bridge between innate immunity and adaptive IgE responses. J Endotoxin Res 7(1):45-8 (2001) PMID 11521081
  268. Hochspringen ↑ Mouse B1 cell line responds to lipopolysaccharide via membrane-bound CD14. J Endotoxin Res 7(1):39-43 (2001) PMID 11521080
  269. Hochspringen ↑ Lipopolyamines as a therapeutic strategy in experimental Gram-negative bacterial sepsis. J Endotoxin Res 7(1):35-8 (2001) PMID 11521079
  270. Hochspringen ↑ Cloning, sequencing, and functional analysis of three glycosyltransferases involved in the biosynthesis of the inner core region of Klebsiella pneumoniae lipopolysaccharide. J Endotoxin Res 7(1):25-33 (2001) PMID 11521078
  271. Hochspringen ↑ Lipopolysaccharide biosynthesis: which steps do bacteria need to survive? J Endotoxin Res 7(1):3-23 (2001) PMID 11521077
  272. Hochspringen ↑ MD-2 binds to bacterial lipopolysaccharide. J Endotoxin Res 6(6):489-91 (2000) PMID 11521076
  273. Hochspringen ↑ Cytosolic protein ubiquitylation in normal and endotoxin stimulated human peripheral blood mononuclear cells. J Endotoxin Res 6(6):483-8 (2000) PMID 11521075
  274. Hochspringen ↑ MALP-2, a Mycoplasma lipopeptide with classical endotoxic properties: end of an era of LPS monopoly? J Endotoxin Res 6(6):471-6 (2000) PMID 11521073
  275. Hochspringen ↑ Clinically-oriented therapies in sepsis: a review. J Endotoxin Res 6(6):463-9 (2000) PMID 11521072
  276. Hochspringen ↑ Molecular mechanisms of NF-kappaB activation induced by bacterial lipopolysaccharide through Toll-like receptors. J Endotoxin Res 6(6):453-7 (2000) PMID 11521070
  277. Hochspringen ↑ Examination of chlorpromazine and other amphipathic drugs on the activity of lipopolysaccharide antagonists, E5564 and E5531. J Endotoxin Res 6(6):447-52 (2000) PMID 11521069
  278. Hochspringen ↑ Cellular activating properties and morphology of membrane-bound and purified meningococcal lipopolysaccharide. J Endotoxin Res 6(6):437-45 (2000) PMID 11521068
  279. Hochspringen ↑ The lipemia of sepsis: triglyceride-rich lipoproteins as agents of innate immunity. J Endotoxin Res 6(6):421-30 (2000) PMID 11521066
  280. Hochspringen ↑ Differential roles of TLR2 and TLR4 in the host response to Gram-negative bacteria: lessons from a lipopolysaccharide-deficient mutant of Neisseria meningitidis. J Endotoxin Res 6(5):411-5 (2000) PMID 11521065
  281. Hochspringen ↑ Structural requirements of muramylpeptides for induction of Toll-like receptor 2-mediated NF-kappaB activation in CHO cells. J Endotoxin Res 6(5):407-10 (2000) PMID 11521064
  282. Hochspringen ↑ Role of MD-2 in TLR2- and TLR4-mediated recognition of Gram-negative and Gram-positive bacteria and activation of chemokine genes. J Endotoxin Res 6(5):401-5 (2000) PMID 11521063
  283. Hochspringen ↑ A horseshoe crab receptor structurally related to Drosophila Toll. J Endotoxin Res 6(5):397-9 (2000) PMID 11521062
  284. Hochspringen ↑ Co-operative induction of pro-inflammatory signaling by Toll-like receptors. J Endotoxin Res 6(5):393-6 (2000) PMID 11521061
  285. Hochspringen ↑ Innate recognition of lipopolysaccharide by Toll-like receptor 4/MD-2 and RP105/MD-1. J Endotoxin Res 6(5):389-91 (2000) PMID 11521060
  286. Hochspringen ↑ The role of Toll-like receptors and MyD88 in innate immune responses. J Endotoxin Res 6(5):383-7 (2000) PMID 11521059
  287. Hochspringen ↑ Phase variation of Coxiella burnetii strain Priscilla: influence of this phenomenon on biochemical features of its lipopolysaccharide. J Endotoxin Res 6(5):369-76 (2000) PMID 11521057
  288. Hochspringen ↑ A monoclonal antibody recognizing the 3-deoxy-D-manno-oct-2-ulosonic acid (Kdo) trisaccharide alphaKdo(2-->4)alphaKdo(2-->4)alphaKdo of Chlamydophila psittaci 6BC lipopolysaccharide. J Endotoxin Res 6(5):361-8 (2000) PMID 11521056
  289. Hochspringen ↑ Lipopolysaccharides in the development of the Guillain-Barre syndrome and Miller Fisher syndrome forms of acute inflammatory peripheral neuropathies. J Endotoxin Res 6(5):341-59 (2000) PMID 11521055
  290. Hochspringen ↑ Induction of TNF-alpha and MnSOD by endotoxin: effect of E5531, a synthetic non-toxic lipid A analog. J Endotoxin Res 6(4):329-35 (2000) PMID 11521054
  291. Hochspringen ↑ Endotoxin tolerance protects against local hepatic ischemia/reperfusion injury in the rat. J Endotoxin Res 6(4):321-8 (2000) PMID 11521053
  292. Hochspringen ↑ Bacterial endotoxin (lipopolysaccharide) stimulates the rate of iron oxidation. J Endotoxin Res 6(4):313-9 (2000) PMID 11521052
  293. Hochspringen ↑ Signaling pathways involved in lipopolysaccharide stimulation of prostaglandin production by rat hypothalamic astroglial cells. J Endotoxin Res 6(4):307-11 (2000) PMID 11521051
  294. Hochspringen ↑ The search for Lps: 1993-1998. J Endotoxin Res 6(4):269-93 (2000) PMID 11521047
  295. Hochspringen ↑ 6th Conference of the International Endotoxin Society. Paris, 24-27 August, 2000. Abstracts. J Endotoxin Res 6(2):73-198 (2000) PMID 11187002
  296. Hochspringen ↑ Synthesis of neoglycoproteins containing Kdo epitopes specific for Chlamydophila psittaci lipopolysaccharide. J Endotoxin Res 6(1):57-69 (2000) PMID 11061033
  297. Hochspringen ↑ Limits of a deletion spanning Tlr4 in C57BL/10ScCr mice. J Endotoxin Res 6(1):51-6 (2000) PMID 11061032
  298. Hochspringen ↑ Endotoxin-induced alterations of lipid and fatty acid compositions in rat liver peroxisomes. J Endotoxin Res 6(1):41-50 (2000) PMID 11061031
  299. Hochspringen ↑ Lipopolysaccharide (LPS)-induced cell death of C3H mouse peritoneal macrophages in the presence of cycloheximide: different susceptibilities of C3H/HeN and C3H/HeJ mice macrophages. J Endotoxin Res 6(1):33-9 (2000) PMID 11061030
  300. Hochspringen ↑ A comparative trial of imipenem versus ceftazidime in the release of endotoxin and cytokine generation in patients with gram-negative urosepsis. Urosepsis Study Group. J Endotoxin Res 6(1):25-31 (2000) PMID 11061029
  301. Hochspringen ↑ Reversible binding of heparin to the loop peptide of endotoxin neutralizing protein. J Endotoxin Res 6(1):17-23 (2000) PMID 11061028
  302. Hochspringen ↑ Luminescence studies of the phagocyte response to endotoxin infusion into normal human subjects: multiple discriminant analysis of luminescence response and correlation with phagocyte morphologic changes and release of elastase. J Endotoxin Res 6(1):3-15 (2000) PMID 11061027
  303. Hochspringen ↑ Endotoxin-mimetic effect of antibodies against Toll-like receptor 4. J Endotoxin Res 6(3):257-64 (2000) PMID 11052181
  304. Hochspringen ↑ The role of polar and facial amphipathic character in determining lipopolysaccharide-binding properties in synthetic cationic peptides. J Endotoxin Res 6(3):249-56 (2000) PMID 11052180
  305. Hochspringen ↑ Complement activation by Proteus mirabilis negatively charged lipopolysaccharides. J Endotoxin Res 6(3):223-34 (2000) PMID 11052177
  306. Hochspringen ↑ LPS and LAM activation of the U373 astrocytoma cell line: differential requirement for CD14. J Endotoxin Res 6(3):215-22 (2000) PMID 11052176
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